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Studies have revealed that metabolic reprogramming, mediated by key metabolic enzymes (including mTOR, HIF-1, and AMPK) and pathways (such as glycolysis and lipid metabolism), acts as a major regulator of Th17/Treg differentiation and function owing to their distinct metabolic profiles
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Fertility, pregnancy, and fetal development-specific data are extremely limited, so reproductive use is extrapolated from other human data and animal studies
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