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Cleavage of SDF-1 by DPP-4 results in loss of activity (65), and consistent with this, DPP-4 inhibition in humans has been associated with enhancement of some effects which are associated with SDF-1 [increased levels of circulating endothelial progenitor cells (66) and increased distal tube sodium excretion (64)]

Researchers also point out that a seven-amino acid synthetic peptide based on the actin-binding part of TB4 was able to promote repair in the aged animals comparable to that observed with the parent molecule. The amino acid sequence noted was LKKTETQ TB-500 without the N-terminal acetylation.26 November 2011 review In a review of TB4s basic properties and clinical applications, researchers dub it a regenerative peptide due to its role in repairing injured cells and tissues

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