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Since GSTO1 has been known to cause de-glutathionylation of -actin impeding its polymerization into filamentous actin 11 , such a downregulation of molecules involved in cytoskeleton organization and adhesion is particularly interesting

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All in all, astrocytic responses in AD are complex and disease stage-specific: in the early, compensated phase of AD (manifested by mild cognitive impairment) astrocytes, together with other neuroglia become reactive and arguably protect brain tissue

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