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Nonsteroidal Versus Steroidal Mineralocorticoid Receptor Antagonists in Heart Failure: RealWorld Evidence From a Multicenter Cohort Study

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In summary, GSH enables antibiotic inactivation by serving as a substrate for detoxifying enzymes, such as FosA, and by modulating the expression or stability of antibiotic-degrading enzymes, including the downregulation of NDM-1